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私は現在、神戸大学大学院法医学分野で常勤専任講師として、研究・教育および法医実務(解剖・死因調査)に携わっています。研修医時代に、神戸大学病院の救急科や循環器内科で経験を積んだことから、法医学においても重要なテーマである虚血性心疾患に興味を持ち、現在研究を進めています。主な基礎研究論文→[Leg Med (Tokyo). 2022 Mar;55:102006.]
現在は、ご遺体から得た液体試料や組織から抽出したタンパク質などの物質を分析し、解剖や病理組織検査でも特定が困難な超急性期の虚血性心疾患を死後診断する新しいバイオマーカーの確立を目指しています。また神戸大学精神医学分野と共同で「自殺者の死後サンプルを用いた生物学的研究」を並行して進めています。
法医実務においては、法医学分野で司法解剖や調査法解剖、承諾解剖を、兵庫県監察医務室では非常勤監察医として行政解剖を執刀しています。ここでの経験症例をCase Reportとして積極的に報告しています。
法医学だけで出来る研究は限られているため、他分野との共同研究や連携も積極的に歓迎しています。
また、法医学や死因調査に関する知見を広く社会に還元するため、市民講座や学校教育における講演・解説活動にも積極的に取り組んでいきたいと考えています。
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毎週木曜 13:30〜17:00 (※ただし解剖や出張等で不在の場合があります)【免許・資格等】
医師免許(2016年3月)
死体解剖資格(2020年12月)
日本法医学会法医認定医(2023年4月)【座右の銘】
千思万考、万物斉同
研究活動情報
■ 受賞- 2022年06月 日本法医学会, 優秀賞, Relations between FURIN SNP genotype, expression and coronary atherosclerosis in autopsy specimens
- 2018年03月 神戸大学医学部附属病院, 2017年度ベストレジデント
- 2017年11月 日本循環器学会, 優秀演題賞, 急性増悪の経過をたどった悪性関節リウマチに伴う肺高血圧症の一例
- 2017年10月 日本救急医学会, 優秀演題, 頭部外傷を契機に発症した中枢性塩類喪失症候群に対して,鉱質コルチコイドを使用し軽快した一例
- INTRODUCTION: More than 720 000 individuals die by suicide annually worldwide, making it a major public health concern and a leading cause of death among young people, particularly in Asian populations. Dysregulation of the 5-hydroxytryptamine system has been implicated in suicide risk. The SLC6A4 gene encodes the serotonin transporter, which regulates synaptic 5-hydroxytryptamine levels. Its promoter region contains a 44-base-pair insertion/deletion polymorphism, known as the serotonin-transporter-linked polymorphic region (5-HTTLPR). We examined the association between the 5-HTTLPR polymorphism and suicide death in a Japanese cohort, representing the largest sample from an Asian population to date. METHODS: We included 448 suicide decedents and 361 controls. Genomic DNA was extracted from peripheral blood, and 5-HTTLPR genotypes were determined using PCR. We evaluated genotype- and allele-level associations with suicide. Additionally, we conducted a meta-analysis incorporating previous studies. RESULTS: The distributions of 5-HTTLPR genotypes and alleles differed significantly between suicide decedents and controls (S allele: OR = 1.62, 95% CI = 1.26-2.09, p < 0.001). The associations remained significant in subgroup analyses by sex, age group, and violent methods. Age- and sex-matched analyses yielded consistent results. However, the random-effects meta-analysis did not demonstrate a significant association. CONCLUSION: Our findings indicate a potential association between 5-HTTLPR polymorphisms and suicide death in a Japanese population. Although further research in larger and diverse samples is warranted, these results may contribute to a better understanding of the biological underpinnings of suicide risk.2026年06月, Neuropsychopharmacology reports, 46(2) (2), e70120, 英語, 国際誌研究論文(学術雑誌)
- 2026年02月, Journal of Forensic and Legal Medicine研究論文(学術雑誌)
- 2025年09月, APPLIED SCIENCES-BASEL, 15(19) (19), 英語研究論文(学術雑誌)
- A 50-year-old male was found dead in a park. Postmortem analysis using liquid chromatography-tandem mass spectrometry revealed lemborexant concentrations of 1.651 μg/mL in blood from the right heart, 0.236 μg/mL in the urine, and 58.642 μg/mL in the stomach contents. Based on the autopsy findings and postmortem analyses, the cause of death was identified as acute lemborexant poisoning due to an overdose. Although lemborexant is generally considered safe, its excessive ingestion can be fatal. Since no lethal concentration of lemborexant has been reported, the blood levels in this case can serve as a reference. Despite its widespread clinical use, lemborexant is not detected by the rapid urine drug screening tests currently available in Japanese investigative agencies. Forensic pathologists must be vigilant in order not to overlook acute lemborexant poisoning.2024年09月, Legal medicine (Tokyo, Japan), 71, 102527 - 102527, 英語, 国際誌研究論文(学術雑誌)
- CD31, a transmembrane protein expressed on endothelial and hematopoietic cells, plays important roles in leukocyte trafficking, mechanotransduction, angiogenesis, vascular permeability, and regulation of cellular responsiveness. CD31 immunoreactivity is employed as a sensitive and specific endothelial marker in diagnostic pathology. In this study, CD31 expression in myocardial tissues from deceased patients with ischemic heart disease and a mouse model of acute myocardial infarction were examined by immunohistochemical staining. We examined 24 neutral formalin-fixed, paraffin-embedded myocardial tissue samples obtained within 48 h postmortem from the autopsies of patients who were diagnosed with ischemic heart disease. CD31 expression was observed in vascular endothelial and endocardial cells. In necrotic myocardium, diffusion of CD31 antigen was observed. Elevated CD31 expression was observed around myocardial cells undergoing remodeling, suggesting that endothelial proliferation occurred at these sites. In contrast, fibrotic myocardial foci did not show upregulated CD31 expression. The same CD31 expression characteristics as those observed in the human samples were observed in the mouse model. CD31 immunostaining as an endothelial and microvasculature marker may be a useful complement to conventional staining techniques currently used in the diagnosis of ischemic heart disease, and may allow the timing and process of myocardial remodeling to be analyzed in detail.2022年11月, Legal medicine (Tokyo, Japan), 59, 102127 - 102127, 英語, 国際誌研究論文(学術雑誌)
- A male in his 90 s consulted a doctor because he experienced several days of general fatigue and dyspnea. He was diagnosed with heart failure, and diuretic medications taken for 3 days relieved his symptoms. However, he was found dead on the morning of the fourth day after consultation. He had received a third dose of coronavirus disease 2019 (COVID-19) vaccine approximately 2 weeks before death. An autopsy revealed dissection of the ascending aorta and pericardial hemotamponade. The heart showed a white villous surface, and the pericardium was fibrously thick. Microscopic examination revealed pericarditis with predominantly macrophage and lymphocyte infiltration. These histological findings were compatible with those of post-vaccination myocarditis. To the best of our knowledge, histopathologically proven pericarditis after COVID-19 vaccination has not been reported. In the present case, extended inflammation of the aortic adventitia was a possible cause of aortic wall fragility followed by dissection.2022年09月, Legal medicine (Tokyo, Japan), 59, 102154 - 102154, 英語, 国際誌
- A man in his early 60 s who worked at a waste disposal plant had fallen into the refuse pit and was immediately taken to the emergency department for treatment. After 8 days without recovering consciousness, the man died. Antemortem contrast-enhanced computed tomography at the emergency department indicated Stanford type B/DeBakey type IIIb aortic dissection. The autopsy showed a sharp and transverse intimal tear 0.6 cm in length in the aortic isthmus and fractures in the 5th-6th thoracic vertebrae. No structural abnormalities in arterial walls were noted on histopathological examination. The traumatic aortic dissection induced by falling is rare, compared with vehicle crash. Although the verification process was challenging, the cause of death was ultimately concluded as traumatic aortic dissection due to falling into the refuse pit. The following observations were cited as evidence: (1) the location and feature of the intimal tear, (2) the positional relationship between the impact site and the entry tear, and (3) the circumstance of clash impact onto the "cushion" of accumulated waste in the refuse pit. Inquiries into the cause of death, such as those made in this report, are required to provide detailed information on the circumstances of the accident, postmortem examinations, and careful consideration.2022年09月, Forensic science, medicine, and pathology, 英語, 国際誌研究論文(学術雑誌)
- ABSTRACT: Many deaths caused by methanol occur as a result of intentional suicide attempts or accidental ingestion, and several investigators have quantified methanol and formic acid in blood and organs. However, to the best of our knowledge, no reports have described regional differences in the concentration of methanol in the brain. A man in his 50s drank alcohol that had been deliberately contaminated with methanol by his wife, and he died of multiple-organ failure after 4 days of intensive medical treatment including hemodialysis. On medicolegal autopsy, cross sections of the brain showed scattered petechial hemorrhage in the brain stem and microscopic hemorrhage with congestion in the bilateral putamina, which showed pinkish discoloration. The concentrations of methanol, formic acid, and ethanol in autopsy samples were measured by headspace gas chromatography, revealing relatively high concentrations of residual methanol and formic acid in the brain (especially in the basal ganglia), although methanol had been eliminated from the blood. Even after 4 days of medical treatment, postmortem toxicological analysis of the brain tissue indicated methanol ingestion. The accumulation of formic acid and the consequent local metabolic acidosis may cause brain lesions.2022年08月, The American journal of forensic medicine and pathology, 英語, 国際誌研究論文(学術雑誌)
- 日本法医病理学会, 2022年08月, 法医病理, 28(1) (1), 45 - 50, 日本語大腿動脈バイパス手術後大量出血による急性失血死症例
- BACKGROUND: Coronary artery disease (CAD), including coronary atherosclerosis (CAS), is one of the most common causes of death. The FURIN SNP rs17514846 is assumed to be a risk factor for CAD. We evaluated this relationship using autopsy specimens and autopsy data, such as the histopathological degree of CAS. MATERIALS AND METHODS: A total of 106 samples were genotyped from obtained blood samples. Myocardial and coronary arterial FURIN levels were quantified by ELISA. The degree of CAS was classified histopathologically according to the Stary classification, and the localization of FURIN was examined by immunostaining. The obtained data were analyzed statistically. RESULTS: FURIN expression was widely observed in the myocardium, vascular smooth muscle cells, endothelial cells, adipocytes, and macrophages. FURIN level in the myocardium of cases with the AA genotype at the FURIN SNP rs17514846 was higher than that in CC cases. Additionally, FURIN levels in both coronary arteries and myocardium were higher at the early stage of CAS than at the late stage microscopically. CONCLUSION: Our study suggested that the A allele of rs17514846 is associated with higher FURIN level in the heart and that FURIN exhibits a higher level in the early stage of CAS. These findings deepen our understanding of the mechanism of CAS.2022年03月, Legal medicine (Tokyo, Japan), 55, 102006 - 102006, 英語, 国際誌研究論文(学術雑誌)
- von Willebrand factor (VWF) plays a crucial role in hemostasis and thrombosis. VWF is involved in platelet attachment to the subendothelium, serving as a carrier protein for coagulation factor VIII. In this study, myocardial tissues from deceased patients with ischemic heart disease and a mouse model of acute myocardial infarction were subjected to immunohistochemistry to determine VWF expression. We examined 28 neutral formalin-fixed, paraffin-embedded myocardial tissue samples obtained from the autopsies of patients who were diagnosed with ischemic heart disease within 48 h postmortem. Most myocardial cells were negative for VWF, although some cells showed nonspecific positivity. Elevated VWF expression was observed around myocardial cells undergoing remodeling, suggesting that endothelial proliferation occurred at these sites. In contrast, completely fibrotic myocardial foci did not show upregulated VWF expression. Positivity in fibrin deposition and hemorrhagic sites was observed. The same VWF expression characteristics as those observed in the human samples were observed in the mouse model. VWF immunostaining as an endothelial marker may be a useful supplementation to conventional staining techniques that are currently used in the diagnosis of ischemic heart disease in terms of examining the timing of myocardial remodeling in detail and highlighting the remodeling process.2022年02月, Legal medicine (Tokyo, Japan), 54, 101997 - 101997, 英語, 国際誌研究論文(学術雑誌)
- Vimentin is a type III intermediate filament cytoskeletal protein that is expressed mainly in cells of mesenchymal origin and is involved in a plethora of cellular functions. In this study, myocardial tissues from patients with ischemic heart disease and a mouse model of acute myocardial infarction were subjected to immunohistochemistry for vimentin. We first examined 26 neutral formalin-fixed, paraffin-embedded myocardial tissue samples from autopsies of patients that were diagnosed with ischemic heart disease within 48 h postmortem. Myocardial cells were negative for vimentin, whereas non-myocardial cells, including vascular endothelium, vascular smooth muscle, fibroblasts, nerve fibers, adipocytes and mesothelial cells, showed positivity. Elevated vimentin expression was observed around myocardial cells undergoing remodeling, suggesting fibroblastic and endothelial proliferation in these locations. By contrast, myocardial foci that were completely fibrotic did not show upregulated vimentin expression. Inflammatory foci including macrophages and neutrophils were clearly visualized with vimentin immunostaining. The same vimentin expression phenomena as those found in human samples were observed in the mouse model. Our study indicates that immunostaining of vimentin as a marker for myocardial remodeling and the dynamics of all non-myocardial cell types may be useful for supplementing conventional staining techniques currently used in the diagnosis of ischemic heart disease.2022年02月, Legal medicine (Tokyo, Japan), 54, 102003 - 102003, 英語, 国際誌研究論文(学術雑誌)
- Thrombomodulin is a transmembrane glycoprotein that is ubiquitously expressed on the surface of vascular endothelial cells. Thrombomodulin exerts its anticoagulant effects by combining with thrombin, activating protein C, and inactivating the coagulation factors FVa and FVIIIa. Clinically, thrombomodulin is also known as a marker of vascular injury because it circulates freely in response to endothelial injury. In this study, myocardial tissue from cases of ischemic heart disease was subjected to immunohistochemistry by thrombomodulin. We examined 40 neutral-formalin-fixed, paraffin-embedded myocardial tissue samples from autopsy cases that were diagnosed with ischemic heart disease (within 48 h postmortem). Thrombomodulin expression was observed in vascular endothelial cells between myocardial cells and in mesothelial cells of the epicardium. In necrotic myocardium, diffusion of thrombomodulin, which reflected endothelial injury, was observed. Upregulated thrombomodulin expression was observed around myocardial cells under ongoing remodeling, which suggested endothelial proliferation in these locations. Completed fibrotic foci of the myocardium did not show upregulated thrombomodulin expression. In a mouse model of acute myocardial infarction, the same phenomena as that found in human samples were observed by immunohistochemistry of thrombomodulin. Immunostaining of thrombomodulin, as a marker for endothelial injury or myocardial remodeling, may be useful for supplementing conventional staining techniques in the diagnosis of ischemic heart disease in forensic pathology.2021年07月, Legal medicine (Tokyo, Japan), 51, 101897 - 101897, 英語, 国際誌研究論文(学術雑誌)
- Rosai-Dorfman disease (RDD) is a rare non-Langerhans cell histiocytosis that is characterized histopathologically by accumulation of CD68-positive, S100-positive, and CD1a-negative histiocytes. Cardiac involvement of RDD is rare. We report here an autopsy case of cardiac involvement of RDD presenting as fibrinous pericarditis. A 14-year-old Japanese boy complained of loss of appetite and breathing difficulty when lying down. He was found dead on his back in his bedroom. One year before his death, he was diagnosed with RDD after skin biopsy. At autopsy, the deceased was 153 cm in height and weighed 38 kg with systemic edema. He had flat pigmented light-brown spots, as well as many pale reddish-brown papules on the abdomen and both thighs. Cervical and mediastinal lymphadenopathy was observed. A large amount of pleural and ascitic fluid was observed. The spleen weighed 381.9 g and showed splenomegaly. The heart weighed 620 g and showed acute fibrinous pericarditis with adhesion. Abundant fibrin was observed on the epicardial surface. The infiltrating cells were CD68-positive, S100-positive, and CD1a-negative histiocytes. The skin and spleen showed histiocytic involvement. Systemic edema, large amounts of pleural and ascitic fluid, a high brain natriuretic peptide level in blood, and hemosiderin-laden macrophages in the lungs suggested chronic heart failure. We speculate that the cause of death was extranodal cardiac involvement of RDD with chronic heart failure. This case highlights the need for forensic pathologists to perform a complete autopsy to determine the cause of sudden death when cardiac involvement of RDD is present.2021年02月, Legal medicine (Tokyo, Japan), 48, 101812 - 101812, 英語, 国際誌研究論文(学術雑誌)
- The deceased was a 44-year-old male who was treated for a suspected Ebstein's anomaly observed using transthoracic echocardiogram. He was found dead in his bed at home. Autopsy revealed that the septal tricuspid leaflet was intact; however, a large anterior tricuspid leaflet cleft and right atrioventricular cavity dilation were observed. Pathological examination revealed a normal tricuspid valve, except for the presence of a cleft with local fibrosis of the left ventricle papillary muscle and hemosiderin-containing macrophages at both lungs. There were no other abnormalities that may have led to death. It was concluded that he died a cardiac death based on the right heart overload associated with the anterior tricuspid leaflet cleft. This case indicates the possibility that the anterior tricuspid leaflet cleft can cause death and also highlights the necessity of a detailed autopsy to accurately diagnose the cause of death.2020年03月, The American journal of forensic medicine and pathology, 41(1) (1), 40 - 41, 英語, 国際誌研究論文(学術雑誌)
- A Japanese woman in her 30s was found dead on a mattress. She had had fever, cough, and dyspnea for about 2 weeks. Gross examination at autopsy revealed slight enlargement of the thyroid gland and histopathological examination resulted in a diagnosis of chronic lymphocytic thyroiditis. The concentration of triiodothyronine in the cadaveric blood was extraordinarily high, whereas that of thyroid stimulating hormone was below the detection limit. Autoimmune antibodies against thyroid tissue were positive. The cause of death was assumed to be congestive heart failure caused by thyroid storm associated with chronic lymphocytic thyroiditis. Systemic histopathological examination of tissues and postmortem biochemistry can enable a diagnosis in medicolegal autopsies.2019年09月, Legal medicine (Tokyo, Japan), 44, 101624 - 101624, 英語, 国際誌
- Rheumatoid vasculitis is a rare etiology for pulmonary hypertension (PH) in patients with connective tissue disease. We encountered a case of acute PH crisis in a case with rheumatoid vasculitis eight months after undergoing adalimumab reduction. Since no repetition of arthralgia occurred after the adalimumab reduction, we decided to not increase the dose of adalimumab. However, hemodynamic collapse thereafter developed and even though steroid pulse therapy was administered, the patient nevertheless died. The autopsy showed clusters of acute and chronic inflammation around the remodeled pulmonary arteries along with micro-thrombi in the vessel lumen. We should consider the possibility of critical worsening of PH as a phenotype of vasculitis related to immunosuppressive therapy reduction.2019年, Internal medicine (Tokyo, Japan), 58(4) (4), 593 - 601, 英語, 国内誌研究論文(学術雑誌)
- (株)へるす出版, 2024年07月, 中毒研究, 37(2) (2), 221 - 221, 日本語法医学と中毒 薬物検査結果から死因を判断する
- 日本法医病理学会, 2023年12月, 法医病理, 29(2) (2), 51 - 52, 日本語
- 日本法医病理学会, 2023年01月, 法医病理, 28(2) (2), 78 - 79, 日本語
- 日本法医病理学会, 2022年03月, 法医病理, 27(2) (2), 115 - 115, 日本語
- (株)へるす出版, 2020年12月, 中毒研究, 33(4) (4), 362 - 362, 日本語覚醒剤の経直腸投与による急性中毒死例
- (株)へるす出版, 2020年12月, 中毒研究, 33(4) (4), 362 - 363, 日本語検視時には指摘されなかった覚醒剤中毒症例
- (NPO)日本法医学会, 2020年12月, 日本法医学雑誌, 74(2) (2), 140 - 140, 日本語インフルエンザウイルスA/H1N1pdm罹患児の突然死2症例
- (NPO)日本法医学会, 2020年12月, 日本法医学雑誌, 74(2) (2), 143 - 143, 日本語心アミロイドーシスの2例
- (NPO)日本法医学会, 2020年08月, 日本法医学雑誌, 74(1) (1), 50 - 50, 日本語心筋組織に於ける炎症性蛋白質ペントラキシン3の発現分布に関する検討
- (NPO)日本法医学会, 2020年08月, 日本法医学雑誌, 74(1) (1), 63 - 63, 日本語虚血性心疾患剖検例心筋組織におけるビメンチン発現の免疫組織化学的検討
- (NPO)日本法医学会, 2020年08月, 日本法医学雑誌, 74(1) (1), 66 - 66, 日本語心外膜脂肪組織及び心筋におけるマクロファージの局在と冠動脈硬化との関連性について
- 日本法医病理学会, 2020年01月, 法医病理, 25(2) (2), 74 - 75, 日本語虐待による頭部外傷の約1年半後に死亡し剖検に至った1歳7ヵ月児の一例
- (NPO)日本法医学会, 2019年12月, 日本法医学雑誌, 73(2) (2), 161 - 161, 日本語臨床上エプスタイン病と疑われていた三尖弁異常症の剖検例
- (NPO)日本法医学会, 2019年12月, 日本法医学雑誌, 73(2) (2), 162 - 162, 日本語剖検により診断された甲状腺機能異常症の2症例
- 日本法医病理学会, 2019年07月, 法医病理, 25(1) (1), 21 - 25, 日本語
- (NPO)日本法医学会, 2019年05月, 日本法医学雑誌, 73(1) (1), 67 - 67, 日本語低体温症がHSP70発現とその死後変化に及ぼす影響
- (NPO)日本法医学会, 2019年05月, 日本法医学雑誌, 73(1) (1), 82 - 82, 日本語虚血性心疾患剖検例心筋組織における内皮関連分子の免疫組織化学的検討
- (NPO)日本法医学会, 2019年05月, 日本法医学雑誌, 73(1) (1), 111 - 111, 日本語炎症性蛋白質ペントラキシン3の法医学応用を目指した死後変化に関する基礎的検討
- (一社)日本救急医学会, 2017年09月, 日本救急医学会雑誌, 28(9) (9), 526 - 526, 日本語頭部外傷を契機に発症した中枢性塩類喪失症候群に対して、鉱質コルチコイドを使用し軽快した一例
- 第9回日本法医病理学会学術全国集会, 2026年09月, 日本語心尖部左心室内腔に偽腫瘍性血栓を認めた一解剖事例口頭発表(一般)
- 第110次日本法医学会学術全国集会, 2026年06月, 日本語急性発症1型糖尿病によって死亡した若年者の一剖検例ポスター発表
- 第2回 京阪神法医学ワークショップ, 2026年03月, 日本語虚血性心疾患の新規死後診断に向けた実験研究についてシンポジウム・ワークショップパネル(指名)
- 第46回日本中毒学会西日本地方会, 2026年03月, 日本語, 日本中毒学会, 大阪薬物スクリーニング検査では診断困難なチエノジアゼピン系向精神薬中毒事例口頭発表(一般)
- 第72回日本法医学会学術近畿地方集会, 2025年11月, 日本語尿中薬物スクリーニングキットでは検出されなかった急性エチゾラム中毒死事例口頭発表(一般)
- 第60回日本アルコール・アディクション医学会学術総会, 2025年10月, 日本語, 日本アルコール・アディクション医学会, 東京死亡直前の飲酒状況に及ぼす同居者の影響ポスター発表
- 第109次日本法医学会学術全国集会, 2025年06月, 日本語, 日本法医学会, 久留米神戸大学法医学分野における法医解剖の現状口頭発表(一般)
- 第1回 京阪神法医学ワークショップ, 2025年03月, 日本語神戸大学法医学分野 〜今、何が新しい?〜シンポジウム・ワークショップパネル(指名)
- 第71回日本法医学会学術近畿地方集会, 2024年11月, 日本語神戸市における新型コロナウイルス抗原陽性死亡例のシリーズ報告口頭発表(一般)
- 第7回日本法医病理学会学術全国集会, 2024年09月, 日本語嚢状瘤に伴った上⾏⼤動脈-右気管⽀瘻による急性窒息死の剖検例口頭発表(一般)
- 第70回日本法医学会学術近畿地方集会, 2023年11月, 日本語解剖時に偶発的に発見された組織黒変症の一剖検例口頭発表(一般)
- 第70回日本法医学会学術近畿地方集会, 2023年11月, 日本語冠状動脈硬化症事例におけるNO関連タンパク質の局在口頭発表(一般)
- 第6回日本法医病理学会学術全国集会, 2023年09月, 日本語慢性膵炎による死亡が疑われた若年女性の一剖検例口頭発表(一般)
- 第107次日本法医学会学術全国集会, 2023年06月, 日本語, 日本法医学会, 小田原法医解剖における遺族満足度調査口頭発表(一般)
- 第44回日本法医学会学術中部地方集会/第69回日本法医学会学術近畿地方集会, 2022年10月, 日本語, 日本法医学会, 三重破損した切断用研削盤による頸部切断の一剖検例ポスター発表
- 第5回日本法医病理学会学術全国集会, 2022年09月, 日本語, 日本法医病理学会, 横浜大脳皮質動脈破綻による急性特発性硬膜下血腫が疑われた一剖検例口頭発表(一般)
- 第106次日本法医学会学術全国集会, 2022年06月, 日本語, 日本法医学会, 愛知Relations between FURIN SNP genotype, expression and coronary atherosclerosis in autopsy specimens口頭発表(一般)
- 第68回日本法医学会学術近畿地方集会, 2021年11月, 日本語, 日本法医学会, 大阪大腿動脈バイパス手術後大量出血による急性失血死症例口頭発表(一般)
- 第105次日本法医学会学術全国集会, 2021年09月, 日本語, 日本法医学会, 福岡The association between genetic variation and protein expression of FURIN in human cardiac tissueポスター発表
- 第67回日本法医学会学術近畿地方集会, 2020年11月, 日本語, 日本法医学会, 滋賀外傷性大動脈解離と考えられた墜落死の剖検例口頭発表(一般)
- 第40回日本中毒学会西日本地方会, 2020年02月, 日本語, 日本中毒学会, 兵庫検視時には指摘されなかった覚醒剤中毒症例口頭発表(一般)
- 第66回日本法医学会学術近畿地方集会, 2019年11月, 日本語, 日本法医学会, 大阪インフルエンザウイルスA/H1N1pdm罹患児の突然死2症例口頭発表(一般)
- 第65回日本法医学会学術近畿地方集会, 2018年11月, 日本語, 日本法医学会, 兵庫臨床上エプスタイン病と疑われていた 三尖弁異常症の剖検例口頭発表(一般)
- 第124回日本循環器学会近畿地方会, 2017年11月, 日本語, 日本循環器学会, 大阪急性増悪の経過をたどった悪性関節リウマチに伴う肺高血圧症の一例口頭発表(一般)
- 第45回日本救急医学会総会・学術集会, 2017年10月, 日本語, 日本救急医学会, 大阪頭部外傷を契機に発症した中枢性塩類喪失症候群に対して,鉱質コルチコイドを使用し軽快した一例ポスター発表
- 第114回日本内科学会・講演会 医学生・研修医の日本内科学会ことはじめ2017 東京, 2017年04月, 日本語, 日本内科学会, 東京抗インターフェロンγ抗体が陽性であった播種性非結核性抗酸菌症の一例ポスター発表
- 第214回日本内科学会近畿地方会, 2016年12月, 日本語, 日本内科学会, 大阪Upfront combination therapyに治療抵抗性を認めた長期覚醒剤乱用歴のある肺動脈性肺高血圧症の一例口頭発表(一般)
■ 共同研究・競争的資金等の研究課題
- 日本学術振興会, 科学研究費助成事業, 若手研究, 神戸大学, 2025年04月01日 - 2028年03月31日心嚢液中タンパク質解析を用いた虚血性心疾患死後診断の研究
- 日本学術振興会, 科学研究費助成事業 研究活動スタート支援, 研究活動スタート支援, 神戸大学, 2022年08月31日 - 2024年03月31日ヒト剖検試料を用いたNO関連タンパク質解析による虚血性心疾患死後診断の研究
- その他, 2026年09月 - 現在司法解剖
- その他, 2025年10月 - 現在承諾解剖
- その他, 2023年05月 - 現在調査法解剖
- その他, 2019年04月 - 現在監察医解剖
- 講師, 第47回兵庫県医師会臨床警法医会研修会「屋外での死亡」, 2025年10月11日 - 2025年10月11日死体検案書の記載について
